Much maligned for years as a saturated fat, we’ll bring you truth regarding the incredible health properties of coconut oil.
As Alzheimer’s Drugs Continue to Fail, Researchers Search for Reasons Why Coconut Oil Cures Alzheimer’s
A group of New York City’s Mt. Sinai Medical School researchers set out to analyze why and how coconut oil affects Alzheimer’s Disease (AD) patients favorably. They acknowledged some efficacy from coconut oil, and they wanted to know more. Since most drug trials to develop an Alzheimer's drug have failed miserably, the motivation to study coconut oil in order to understand how it positively affects AD was probably motivated by a desire to patent a new drug that could work similar to coconut oil, a natural food that cannot be patented. Their research was based on earlier animal studies of virgin coconut oil’s effect on mouse models of coconut oil as well as other in vitro (lab culture) studies. The researchers also acknowledged the existence of many anecdotal testimonies of AD (Alzheimer’s disease) patients. The Mt. Sinai report was published in the journal Science Direct, Brain Research with the title: Coconut oil decreases expression of amyloid precursor protein (APP) and secretion of amyloid peptides through inhibition of ADP-ribosylation factor 1 (ARF1).
A new study published in the journal Plant Foods for Human Nutrition on August 31, 2018, with the title Beneficial Effect of Virgin Coconut Oil on Alloxan-Induced Diabetes and Microbiota Composition in Rats adds further proof that virgin coconut oil is a powerful remedy for diabetes. In this latest study, virgin coconut oil was shown to be beneficial to the microbiome by increasing probiotic bacteria, leading to better outcomes for those suffering with diabetes. This new research confirms what many have reported to us over the past 15 plus years, that virgin coconut oil is beneficial in overcoming both Type 1 and Type 2 diabetes. This new study offers a possible explanation as to how virgin coconut oil helps people with Type 1 diabetes without directly affecting insulin levels.
The corporate-sponsored "mainstream" media, funded in large part by the pharmaceutical industry, is promoting a lecture given in German by a Harvard medical professor who claims that coconut oil is "pure poison." The lecture was given by Dr. Karin Michels, the director of the Institute for Prevention and Tumor Epidemiology at the University of Freiburg and a professor at the Harvard T.H. Chan School of Public Health. Now, before I expose the old same tired lies the medical system and its media proponents claim about coconut oil, and the reasons why they are doing so, just consider the logic of making such a statement as "coconut oil is pure poison." While coconut oil has only become popular in the U.S. again in recent years, it is a traditional food that has been consumed in tropical climates where coconuts grow for many thousands of years. Is it reasonable to think that all these billions of people have been poisoning themselves due to their consumption of coconut oil for the past several thousand years? If it were truly a poison leading to heart disease and bad health, wouldn't we be seeing an increase in many health problems among the millions of Americans who now use it as their primary dietary oil? Is there any evidence at all for any of Dr. Michels claims? No. The real reason coconut oil is being attacked in the corporate media by the medical system, is because it is a threat to their toxic drugs. Because outside of the U.S., the peer reviewed literature is publishing more and more studies showing just how healthy coconut oil really is, and how it is being used in nutritional protocols to help cure many diseases such as dementia and Alzheimer's disease.
A new review published in August 2018, in the Journal Brain Sciences looked at the effectiveness of the ketogenic diet to treat adult epilepsy, adult malignant glioma (brain tumors), and Alzheimer’s disease. It was written by Tanya J. W. McDonald and Mackenzie C. Cervenka from the Department of Neurology, Johns Hopkins University School of Medicine. The review is titled "The Expanding Role of Ketogenic Diets in Adult Neurological Disorders." The aim of the review is to describe the evidence, preclinical and clinical, supporting Ketogenic Diet use in the management of adult epilepsy, adult malignant gliomas (brain tumors), and Alzheimer’s disease. Several randomized controlled trials support the use of Ketogenic Diets for the treatment of drug-resistant epilepsy and there is emerging evidence that these diets are also effective in treating refractory status epilepticus, malignant glioma and Alzheimer’s disease in adults.
There have been several studies from countries outside the U.S. that show the health virtues of coconut oil for Alzheimer's Disease. Meanwhile, pronouncements from the USA’s Alzheimer's Association and medical press releases, warning against using coconut oil, have persisted. These international studies conclusions fly in the face of our “expert” opinions that, as a saturated fat, coconut oil threatens heart health. This misinformation continues even as medical practitioners of all types are realizing that the lipid theory of heart disease as the leading contributor to poor heart health is simply not true. The most recent study was published on July 20th, by the Journal of Alzheimer's Disease. This human study was conducted by academic researchers in Valencia, Spain, and titled Improvement of Main Cognitive Functions in Patients with Alzheimer's Disease after Treatment with Coconut Oil Enriched Mediterranean Diet: A Pilot Study. This was a remarkable study, as there were measurable improvements among Alzheimer's patients in 21 days from adding coconut oil to a Mediterranean diet.
Another study has been published here in 2018 looking at the positive effects of virgin coconut oil for Alzheimer's disease. This is the third peer-reviewed study we have covered so far in 2018 looking at the effects of coconut oil on Alzheimer's Disease (AD). This latest study is titled "Possible prophylactic anti-excitotoxic and anti-oxidant effects of virgin coconut oil on aluminium chloride-induced Alzheimer's in rat models." It was published July 13, 2018, in the "Journal of Integrative Neuroscience." The study was conducted by researchers from the Faculty of Medicine at King Abdulaziz University in Saudi Arabia. This year's previous studies on AD and coconut oil were also conducted outside of the U.S. (Japan and Iran.) This study is significant, because it addresses the issue of aluminum toxicity, which has been increasingly linked to AD. (See: Study: High Amounts of Aluminum in Brains of Alzheimer’s Patients.) From the study abstract: "Alzheimer’s disease (AD) is a progressive neurodegenerative disease that affects an estimated 5.4 million people worldwide. However, there remains no curative treatment for the condition. Aβ and hyperphosphorylated tau accumulation are the main hallmarks of the disease; they interfere with glutamate uptake and mediate glutamate excitotoxicity, oxidative stress, inflammation and neurodegeneration. As virgin coconut oil (VCO) is well-known as an antioxidant and anti-inflammatory natural compound, the purpose of the present study was to assess the possible prophylactic effect of VCO on aluminium chloride (AlCl3)- induced AD in rat."
Scottish medical doctor, Malcolm Kendrick, has just written a brilliant expose on his blog explaining, scientifically, why it is impossible for saturated fats to raise LDL cholesterol levels. As I have written many times over the years, this is the kind of information that can save your life and help you make wise dietary choices, but it is information that the U.S. government, Big Pharma, and the corporate-sponsored "mainstream" media cannot afford to publish. Because to do so would be to admit guilt in one of the biggest medical scams of all time: the lipid theory of heart disease. This theory, which has been proven scientifically to be false, has been an economic success for cholesterol-lowering statin medical drugs, the most profitable class of medical drugs all time. This theory also promotes the low-fat diet which encourages consumption of carbohydrates from U.S. subsidized crops, as well as polyunsaturated oil, also derived from U.S. subsidized crops. This theory of heart disease, which condemns cholesterol and saturated fat, has probably been responsible for many millions of people's early deaths and the life-long suffering of autoimmune diseases for an entire generation.
In May, 2018, I wrote about a new study published in Iran that looked at the "neuroprotective effects" effects of virgin coconut oil for Alzheimer's patients. The title of the study is Virgin coconut oil (VCO) by normalizing NLRP3 inflammasome showed potential neuroprotective effects in Amyloid-β induced toxicity and high-fat diet fed rat and was published in the journal Food and Chemical Toxicology. While we have been publishing reports of coconut oil reversing the symptoms of Alzheimer's Disease and other neurological diseases for several years now, we believe this was the first peer-reviewed study to actually look at some of the mechanisms of how coconut oil benefits Alzheimer's patients. Now, a second peer-reviewed study has been published here in 2018 looking at the mechanisms of how coconut oil positively affects Alzheimer's patients. Published in the journal, Neurochemical Research, researchers in Japan examined the effects of lauric acid, the most predominant medium chain fatty acid found in coconut oil, on activated microglia in mice. Coconut oil is nature's richest source of lauric acid at about 50% of its composition, and human breast milk comes in a distant second at around 6% lauric acid. The title of the study is Lauric Acid Alleviates Neuroinflammatory Responses by Activated Microglia: Involvement of the GPR40-Dependent Pathway.
Study: Virgin Coconut Oil Reduces Aβ Plaques, Inflammation, and Oxidative Stress Associated with Alzheimer’s Disease
A study published in August 2018 in the journal Food and Chemical Toxicology confirms what many people have already discovered: coconut oil can improve or even reverse the effects of Alzheimer's Disease. This is believed to be the first peer-reviewed study to examine coconut oil's effect on Alzheimer's Disease. The title of the study is "Virgin coconut oil (VCO) by normalizing NLRP3 inflammasome showed potential neuroprotective effects in Amyloid-β induced toxicity and high-fat diet fed rat." Researchers from Kermanshah University of Medical Sciences and Hamadan University of Medical Sciences in Iran used rats in a laboratory to study "the effects of virgin coconut oil (VCO) on inflammasome and oxidative stress in an Alzheimer's model." Their study found that the rats fed VCO "improved hippocampus histological changes, reduced Aβ plaques and phosphorylated Tau." (Phosphorylated Tau is a biological marker for Alzheimer's Disease.) They concluded that virgin coconut oil "showed potential neuroprotective effects." From the study conclusion: "Virgin Coconut Oil improves the health of the hippocampus and improves memory and learning in Alzheimer and HFD model rats by inhibiting inflammasome and reducing oxidative stress."
With the rise of antibiotic resistant pathogens, researchers are turning their attention to natural products in hopes of finding cures outside of the patented pharmaceutical paradigm for fighting disease. In a study just published in Journal of Applied Microbiology, Australian researchers studied 22 natural products and their ability to inhibit the spore cycle of Clostridium difficile. Clostridium difficile (C. diff) is mostly incubated in hospital settings, is antibiotic resistant, and very contagious. It can create life-threatening inflammation of the colon. The Australian researchers tested 22 natural products in vitro, and found that three of them "showed inhibitory effects on sporulation of C. difficile. Effects on sporulation, determined using microscopy and a conventional spore recovery assay, showed that fresh onion bulb extract (6.3% v v-1 ) and coconut oil (8% v v-1 ) inhibited sporulation in all four isolates by 66-86% and 51-88%, respectively, compared to untreated controls. Fresh ginger rhizome extract (25% v v-1 ) was also inhibitory, although to a lesser extent."