There have been two recent animal studies in Spain indicating that beer is capable of removing aluminum from brain cells, allowing the brain to heal from its toxicity and inflammation. The most recent study used non-alcoholic beer, and the earlier one used beer with alcohol. Both types of beer, mostly due to their antioxidant hops content and silica from silicic acid used in the brewing process, have been observed leaching aluminum from aluminum nitrate induced into lab rats. Aluminum that gets into brain tissue creates all sorts of neurological ills. Silica binds with aluminum to neutralize its toxicity and escort it out of the body through urine. Cucumbers and the herb horsetail are high in silica that doesn't penetrate the blood-brain barrier. Beer contains the silicic acid which does. But there is a serious caveat for using beer to eliminate aluminum from the brain that will be discussed in this article.
Another study has been published here in 2018 looking at the positive effects of virgin coconut oil for Alzheimer's disease. This is the third peer-reviewed study we have covered so far in 2018 looking at the effects of coconut oil on Alzheimer's Disease (AD). This latest study is titled "Possible prophylactic anti-excitotoxic and anti-oxidant effects of virgin coconut oil on aluminium chloride-induced Alzheimer's in rat models." It was published July 13, 2018, in the "Journal of Integrative Neuroscience." The study was conducted by researchers from the Faculty of Medicine at King Abdulaziz University in Saudi Arabia. This year's previous studies on AD and coconut oil were also conducted outside of the U.S. (Japan and Iran.) This study is significant, because it addresses the issue of aluminum toxicity, which has been increasingly linked to AD. (See: Study: High Amounts of Aluminum in Brains of Alzheimer’s Patients.) From the study abstract: "Alzheimer’s disease (AD) is a progressive neurodegenerative disease that affects an estimated 5.4 million people worldwide. However, there remains no curative treatment for the condition. Aβ and hyperphosphorylated tau accumulation are the main hallmarks of the disease; they interfere with glutamate uptake and mediate glutamate excitotoxicity, oxidative stress, inflammation and neurodegeneration. As virgin coconut oil (VCO) is well-known as an antioxidant and anti-inflammatory natural compound, the purpose of the present study was to assess the possible prophylactic effect of VCO on aluminium chloride (AlCl3)- induced AD in rat."