by Michael Belkin
The Refusers
Here we have a study pointing out the unintended consequences of vaccination. ‘Antigenic drift occurs when pressure from the body’s immune system causes a virus used in a vaccine to mutate into a slightly different form that can potentially be more infectious.’ In other words, the vaccine creates a more virulent virus.
Overuse of vaccines creates the same problem as the overuse of antibiotics, which creates deadly antibiotic-resistant germs like Methicillin-resistant Staphylococcus aureus (MRSA).
Vaccine fundamentalists see antigenic drift as an opportunity to make more vaccines. The Refusers see antigenic drift as the epitome of pseudo-scientific quackery. Germs are more intelligent and adaptable than vaccine scientists. Doctors mass-inoculate everyone against one disease strain and the bug evolves into another more dangerous strain not covered by the vaccine. So they make a new vaccine against that new strain and then it happens again.
Contrast that transitory, pseudo-scientific immunity with permanent natural immunity acquired by exposure to wild disease strains (actual herd immunity), which vaccine authorities are intent on eliminating through so-called disease eradication. They have bastardized the concept of natural herd immunity to sugar-coat their ineffective vaccination programs.
Vaccine makers see antigenic drift as a profit center: More boosters required for new, more dangerous germs created by their last ineffective vaccine. Then they demonize educated parents who refuse their toxic vaccines by claiming vaccine refusal undermines their perverted version of herd immunity. Quite a racket, isn’t it?
**********************************************************************************************
Researchers reveal the mechanism behind antigenic drift VaccineNewsDaily.com
by Ted Purlain on December 28, 2011
A new study by researchers from the Massachusetts Institute of Technology has revealed the mechanism behind the phenomenon known as antigenic drift.
Antigenic drift occurs when pressure from the body’s immune system causes a virus used in a vaccine to mutate into a slightly different form that can potentially be more infectious.
The scientists, led by Ram Sasisekharan, a professor of health sciences and biological engineering at MIT, conducted the study by examining the chain of amino acids in the viral protein hemagglutinin. They identified which amino acids were most likely to mutate into forms that would improve the viruses’ capability to infect new hosts.
The knowledge acquired by Sasisekharan and his team could help influenza vaccine designers develop vaccines that do not produce fitter viruses, as the evolved strains are known.
New strains of influenza emerge constantly, leaving researchers searching for which new strains should be included in the seasonal influenza vaccine, which must be reformulated every year.
The vaccines stimulate the production of antibodies that target a section of the hemagglutinin protein known as the antigenic site. When a virus encounters antibodies it can change slightly into a form that can spread more easily to those that have not been vaccinated and can bind more tightly to the surfaces of cells in the respiratory tract of flu victims, making it more infectious.
With funding from the U.S. National Institutes of Health and the Singapore MIT Alliance for Research and Technology, Sasisekharan and his team sought to find out how this phenomenon occurs.
They examined the hemagglutinin protein using an approach called network analysis, which looks at the relationship between the individual amino acids that make up the protein. The resulting model showed that those amino acids located in the protein’s antigenic region that were highly linked to those in the receptor-binding region were more likely to change affinity upon mutation. Selection pressure due to vaccination could contribute to the evolution of fitter viruses by producing better-binding hemagglutinin proteins.
“The idea that hemagglutinin can only accommodate certain mutations without losing fitness is not a new one, but what this paper gives us is a way to understand how changes in distant amino acids affect receptor binding,” David Topham, a professor at the University of Rochester School of Medicine, said.
Sasisekharan said that with knowledge of which amino acids are most likely to mutate into a more infectious form, vaccine producers could create vaccines that do not provoke mutations.
“This understanding of the relationship between the antigenic site and the receptor-binding site could be added to the current methods of vaccine selection and vaccine designs to limit drift,” Sasisekharan said.
Full Blog Post on The Refusers: http://therefusers.com/refusers-newsroom/vaccines-create-deadlier-viruses-through-antigenic-drift/
Vaccine Epidemic
How Corporate Greed, Biased Science, and Coercive Government Threaten Our Human Rights, Our Health, and Our Children
by Louise Kuo Habakus and Mary Holland J.D.
FREE Shipping Available!
More Info